The truth about statin drugs

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  1. Matt’s avatar

    Not being a big fan of Statins i would suggest reading these articles.

    Magnesium and Walking Will Always be Superior to Lipitor
    Lifestyle changes that allow you to avoid lipitor

  2. Marc Marshall, D.C.’s avatar

    We should also keep in mind that statins interfere with co-enzyme Q10 production. CoQ10 is extremely important for mitochondrial energy production and is especially important for heart function. It is also a powerful antioxidant which may prevent arteriosclerosis by preventing the accumulation of oxidized fats. It has been shown in clinical studies to modulate high blood pressure, regulate heart rhythm, and increase exercise tolerance in cases of angina and congestive heart failure.

  3. Chris’s avatar

    Hi Marc,

    Welcome to The Healthy Skeptic and thanks for your comment.

    Yes, that’s exactly right. Statins powerfully inhibit CoQ10 production and that is very likely one reason are not effective for more than 95% of the population and actually increase total mortality in many cases – in spite of their anti-inflammatory effect.

    This is one of the many “untold stories” of statin drugs. Thanks for bringing it to our attention!


  4. Micki Jacobs’s avatar

    Aspirin is a vitamin K antagonist. This means that the low dose aspirin that is so often recommended is preventing vitamin K from ensuring strong, flexible arteries with little to no calcium deposition – which is probably what REALLY causes CVD. Not to mention that aspirin ups the risk of bleeding out.
    Aspirin on a daily basis is ill-conceived and is based on the paradigm of CVD occurring as a result platelets sticking together and then hanging up on build-up in arteries. Instead, it appears that calcium build-up is the first action of problems which occurs throughout life from early on – due to insufficient K2 and insufficiency of other calcium metabolism co-factors – and then the build up causes cholesterol to try to repair this problem and then docs think they need to “thin” blood and reduce cholesterol carrier proteins (LDL and VLDL) and they block the vitamin K action of making calcium go to the teeth and bones by hampering the carrier protein of vitamin K (yep, LDL and VLDL are the carrier proteins of vitamin K and beta carotene, among other beneficial proteins) via statins and aspirin. Hence, we have the wrong paradigm, the wrong treatments, and we need less aberrant forms of calcium, more calcium metabolism co-factors and fewer docs prescribing.

  5. Mike, RPh’s avatar

    The comment by Micki Jacobs is incorrect.

    Aspirin is NOT in any way a vitamin K antagonist.  Aspirin is not an anticoagulant like warfarin.  Aspirin is an antiplatelet drug.  It is an irreversible inhibitor of cyclooxygenase, which is an ezyme involved in the production of prostaglandins and, important for this topic, thromboxane.

    Thromboxane causes platelets to clump together.  This clumping together to repair damage blood vessels is often what leads to heart attacks and strokes.  Therefore, Aspirin works to reduce the incidence of stroke and heart attack by blocking the ability of platelets to clump together.

    That’s not to say that low-dose Aspirin does not have it’s own potential adverse effects.  Even low doses of Aspirin can greatly increase one’s risk for gastrointestinal ulcers.  However, everything the aforementioned comment mentioned about it’s effect on Vitamin K is completely false.

    Aspirin has absolutely no effect on Vitamin K.

  6. abolderwoman’s avatar

    Thanks for this post and comments regarding CoQ10, which most physicians seem to ignore. There’s a very good book related to this discussion — “Drug-Induced Nutrient Depletion Handbook.”  It’s not as up-to-date as some of us would like, but it’s still helpful for anyone wondering about how specific drugs can affect our health.   (fyi, I have no connection to this book or its authors — just find it useful and thought others might, too.)

  7. admin’s avatar

    Thanks for the heads-up on that book, abolderwoman.  Actually I just received an email a couple of days ago from a woman who suffered from muscle damage as a result of taking statins.  She was wondering what she can do to address the persistent weakness she’s experiencing – even though she is no longer taking the drugs.  I’ll forward this to her.

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